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Fig. 6 | Molecular Brain

Fig. 6

From: Analysis of neurexin-neuroligin complexes supports an isoform-specific role for beta-neurexin-1 dysfunction in a mouse model of autism

Fig. 6

Association of mutant beta-Nrxn1ΔC with Nlgn1 initiates early after expression. HA-immunoprecipitates from the cortex of control and beta-Nrxn1ΔC mice left untreated (off DOX) or treated with DOX for 14 days and allowed to re-express the mutant beta-Nrxn1ΔC protein for 30 days (on/off DOX) were incubated with antibodies against HA, Nlgn1, Nlgn2, Nlgn3 and LRRTM2, as indicated. Nlgn1 is selectively recovered in beta-Nrxn1ΔC immunoprecipitates

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Molecular Brain

ISSN: 1756-6606